Herz-Kreislaufnetz im NGFN
Deutsche Version Normal View

External links may lead to non-barrier-free websites
Home Hypertension and cardiovascular end organ damageCoronary artery disease and myocardial infarctionCauses, pathways and modifiers of cardiomoypathiesHeart failureHemostaseology / AtherosclerosisInflammation and immune response in the pathogenesis of typ 2 diabetes and atherosclerosis: Testing the common soil hypothesisGenetyp-phenotype association studies in clinically well characterized patients with inherited bleeding disordersVon Willebrand Factor Net projectsOrganigramHot news Locations ContactLinksImprintSponsors Picture of the 1st Network Meeting of the Herz-Kreislauf-NetzDownloadsArchivExternal job opportunities





Recent experimental and clinical data show that cardiac diseases cannot be seen completely apart from the vascular system. The heart and the vascular system are connected by a mutual process of regulation and also pathophysiology. If one system fals the other will be affected as well earlier or later. We know from large experimental studies, analyses from animal models, epidemiologic studies as well as clinical trials that atherosclerosis is a local unspecific inflammatory process which is accompanied by systemic reactions and finally also involves the heart itself. The causes are the coworking of numerous local and systemic atherothrombogenic factors. The same is valid for disturbances of hemostaseology because of their direct effect on thrombogenesis and therefore on myocardial infarction and indirectly on atherosclerotic processes.